Clinical Implications of Basic Research
Fragile X Syndrome: Keys to the Molecular Genetics of Synaptic Plasticity

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    As with general psychiatry, there has never been a better time to be a child psychiatrist. The ability to understand our patients' mental illness and to offer effective treatments is being fed by explosive advances in molecular medicine, proteinomics, neuroimaging, gene mapping, brain mapping, pharmacogenetics, and the identification of genetic polymorphisms and endophenotypes.2–6 ( See the article by Solomon and colleagues elsewhere in this issue for further discussion of this topic.)

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    These findings have opened the door to potential neuropharmacological treatments for fragile X syndrome and may eventually guide us to treatments for mental retardation and autism. In the previous column, we reviewed the genetics of fragile X syndrome.3 Recall that fragile X results from a mutation in the FMR1 gene, which encodes the fragile X mental retardation protein (FMRP).

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