Impulsivity and alcohol use coping motives in a trauma-exposed sample: The mediating role of distress tolerance

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Abstract

The present investigation examined the mediating role of distress tolerance in the association between impulsivity and alcohol use coping motives among trauma-exposed individuals. Participants were 86 adults (64.3% women; Mage = 23.4, SD = 9.3) who met the DSM-IV-TR posttraumatic stress disorder (PTSD) Criterion A for atleast one traumatic life event and endorsed alcohol use in the past month. Distress tolerance at least partially mediated the association between impulsivity and alcohol use coping motives, after controlling for the variance explained by PTSD symptom severity and alcohol use problems. Clinical implications and future directions related to this line of inquiry are presented and discussed.

Introduction

Significant associations have been documented between posttraumatic stress disorder (PTSD) symptoms, including PTSD diagnosis, and alcohol use disorders (e.g., McFarlane et al., 2009, Stewart, 1996). Recent efforts have focused on elucidating the mechanisms underlying the co-occurrence of these clinical problems. The examination of motivations for alcohol use is promising in understanding the association between PTSD and problematic alcohol use. Coping motives, in particular, are significantly related to alcohol consumption and alcohol use problems (Cooper, 1994). Emerging research has indicated that trauma-exposed individuals with or without PTSD are especially likely to demonstrate enhanced motivation to drink alcohol to cope with negative affective states (e.g., Dixon, Leen-Feldner, Ham, Feldner, & Lewis, 2009), and drinking to cope has been shown to mediate the relationship between alcohol consumption rate and PTSD symptoms (Kaysen et al., 2007). Given the associations between PTSD symptoms and alcohol use coping motives, it is pertinent to improve our understanding of cognitive-affective factors that may underlie this relationship to inform clinical interventions. Impulsivity and distress tolerance are two promising factors in this domain, as both possess associations to PTSD symptoms and problem alcohol use.

Impulsivity, the tendency to opt for immediate reward regardless of long-term consequences (Moeller, Barratt, Dougherty, Schmitz, & Swann, 2001), is associated with PTSD (Casada & Roache, 2005), binge drinking (James & Taylor, 2007), and shorter duration of alcohol abstinence (Charney, Zikos, & Gill, 2010). Impulsivity may function as a risk and maintenance factor for PTSD symptoms. Specifically, greater impulsivity may predispose individuals to engage in high-risk behaviors (e.g., substance use), which may lead to increased risk for trauma exposure (e.g., Stewart & Israeli, 2002). Moreover, greater impulsivity post-trauma theoretically may predispose individuals with PTSD symptoms to act upon their symptoms in a risky manner to alleviate the associated distress. Finally, theory and emerging empirical evidence suggest that individuals high in certain forms of impulsivity (e.g., “negative urgency,” or the tendency to “act rashly in response to distress,” Cyders et al., 2007) may more immediately select negative reinforcement opportunities, such as coping-oriented alcohol use, when faced with high levels of negative affect (Cyders & Smith, 2008).

Distress tolerance, the perceived or actual ability to withstand aversive physical or emotional stimuli (Simons & Gaher, 2005), is another promising explanatory factor in the association between PTSD and alcohol use coping motives. Distress tolerance is a theoretically malleable factor (e.g., Linehan, 1993) that has been inversely related to PTSD symptoms (Marshall-Berenz, Vujanovic, Bonn-Miller, Bernstein, & Zvolensky, 2010), alcohol use problems (Buckner, Keough, & Schmidt, 2007), duration of substance use abstinence attempts (Daughters, Lejuez, Kahler, Strong, & Brown, 2005), and coping motives for alcohol use (Buckner et al., 2007). Trauma-exposed individuals with lower levels of distress tolerance may perceive their abilities to withstand emotional distress as compromised, and may be more motivated to use alcohol to regulate negative mood states. Consistent with this perspective, recent work has demonstrated that lower levels of distress tolerance partially mediated the association between PTSD symptoms and alcohol use coping motives among trauma-exposed adults (Vujanovic, Marshall-Berenz, & Zvolensky, in press).

To our knowledge, no studies have examined the association between impulsivity and distress tolerance, although extant models of impulsivity hypothesize key relationships between these constructs (Cyders & Smith, 2008). Further, no empirical work to date has examined the relationships among impulsivity, distress tolerance, and alcohol use coping motives among trauma-exposed adults in one overarching model. Among trauma-exposed individuals, lower levels of perceived distress tolerance (i.e., cognitive self-appraisals of one’s ability to tolerate distress) may help to explain the association between impulsivity and the tendency to opt for negative reinforcement (e.g., alcohol use) without regard for long-term consequences (Cyders & Smith, 2008). In so doing, these individuals may develop or maintain the perception that they cannot tolerate negative affective states without engaging in a behavior to alleviate those states. Lower perceived distress tolerance may thus partially mediate the relationship between impulsivity and coping motives for alcohol use, such that impulsive individuals may experience increased motivation to drink to cope by way of learning that they cannot tolerate episodes of negative affect but rather require immediate relief of the distressing affective state.

The aim of the current study was to investigate the mediating role of perceived distress tolerance in the relationship between self-reported impulsivity and coping-oriented alcohol use motives in a trauma-exposed community sample. Consistent with formal tests of mediation, it was hypothesized that: (1) impulsivity would be positively associated with alcohol use coping motives; (2) impulsivity would be negatively associated with distress tolerance; and (3) distress tolerance would mediate the relationship between impulsivity and alcohol use coping motives (please see Fig. 1). All proposed effects were examined after accounting for the variance contributed by alcohol use problems and PTSD symptom severity.

Section snippets

Participants

Participants were 86 adults (64.3% women; Mage = 23.4, SD = 9.3) who met the Diagnostic and Statistical Manual-IV(DSM-IV-TR)PTSD Criterion A1 and Criterion A2 (American Psychiatric Association [APA], 2000, p. 467) for at least one traumatic life event. Consistent with the Vermont state population (State of Vermont Department of Health, 2007), approximately 94.0% of the sample identified as White/Caucasian, 2.4% as Hispanic/Latino, 1.2% as Asian, 1.2% as Biracial, and 1.2% as “other”. With regard to

Sample characteristics

46.4% of the sample met criteria for one or more current (past month) axis I disorders. Participants with current psychopathology met criteria for an average of 1.05 (SD = 1.57) diagnoses. 16.7% of the total sample met current (past month) criteria for Social Anxiety Disorder, 15.5% for Generalized Anxiety Disorder, 9.5% for Specific Phobia, 9.5% for Dysthymia, 8.3% for Major Depressive Disorder, 6.0% for Obsessive Compulsive Disorder, 4.8% for PTSD [assessed by the CAPS], 2.4% for Eating

Discussion

The present investigation evaluated the mediating role of distress tolerance in the association between impulsivity and alcohol use coping motives. Findings were consistent with hypotheses. First, impulsivity evidenced a significant positive association with alcohol use coping motives, even after accounting for the variance contributed by PTSD symptom severity and alcohol use problems. Although the effect size was small, this finding extends past work documenting significant associations

Acknowledgments

This research was supported by a National Institute on Mental Health National Research Service Award (1 F31 MH080453-01A1) awarded to Erin C. Marshall-Berenz. The authors acknowledge the support of Dr. Michael J. Zvolensky, Ph.D., director of the Anxiety and Health Research Laboratory (AHRL) at the University of Vermont. The data presented in the manuscript were collected in the AHRL under direct supervision of Dr. Zvolensky. The views expressed here reflect those of the authors and do not

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