Coronary and cerebrovascular atherosclerosis: two aspects of the same disease or two different pathologies?
References (34)
- et al.
The geographic pathology of atherosclerosis: a review of the literature with some personal observations on cerebral atherosclerosis
J. Chron. Dis.
(1967) - et al.
The Canadian American Ticlopidine Study (CATS) in Thromboembolic stroke
Lancet
(1989) The cardiovascular pathology of smoking
Am. Heart. J.
(1988)- et al.
Relative protection from cerebral atherosclerosis of young patients with homozygous familial hypercholesterolemia
Atherosclerosis
(1991) - et al.
A nationwide study of atherosclerosis in infants, children and young adults in Japan
Atherosclerosis
(1988) - et al.
Hypertension and cerebral atherosclerosis
Circulation
(1969) Transient cerebral ischemia: pathogenesis, prognosis and management
Ann. R. Coll. Phys. Surg. Can.
(1974)- et al.
Risk factors for atherosclerotic vascular diseases in People's Republic of China
- et al.
Risk factors and concomitants of internal carotid artery occlusion or stenosis: a controlled study of 159 cases
Arch. Neurol.
(1985) - et al.
Cardiovascular determinants of carotid artery disease. The Rotterdam Elderly Study
Hypertension
(1992)
Incrementi dei livelli sierici di IgA e C4 nell'aterosclerosi: assenza di correlazione con il quadro arteriografico
Cardiologia
Race, sex and occlusive cerebrovascular disease: a review
Stroke
La mortalità in Italia nell'anno 1989
Accuracy of death certification of stroke. The Framingham Study
Stroke
Cerebrovascular accidents in Nigerians: a review of 205 cases
W. Afr. Med. J.
Why the American decline in coronary heart disease?
Lancet
Plaque rupture with severe pre-existing stenosis precipitating coronary thrombosis: characteristics of coronary atherosclerotic plaques underlying fatal occlusive thrombi
Br. Heart J.
Cited by (15)
Management of Acute Ischemic Thrombosis
2018, Neurosurgery Clinics of North AmericaCitation Excerpt :LDL oxidation leads to incorporation of cholesterol deposits into the subendothelial layer of the blood vessels. Injury to the intimal layer from accumulation of these deposits can cause platelet aggregation and thrombus formation that may lead to stenosis of the vessel.3 Rupture of plaques can lead to acute ischemic events as well as occlusion at the site of the stenosis.
Remnant-Like Particle Cholesterol, Low-Density Lipoprotein Triglycerides, and Incident Cardiovascular Disease
2018, Journal of the American College of CardiologyCitation Excerpt :Arterial disease may differ among vascular beds, particularly smaller arteries and arterioles (33). Plaque composition in the smaller cerebral arteries suggests a more fibrotic process than in the coronary arteries, which have more lipid-rich cores and typical atheromatous lesions (34). In addition, arteriolar lesions are characterized by hyalinosis instead of lipid.
Endovascular Treatment of Acute Stroke and Occlusive Cerebrovascular Disease
2018, Principles of Neurological SurgeryTherapeutic approaches to drug targets in atherosclerosis
2014, Saudi Pharmaceutical JournalCitation Excerpt :Atherosclerosis progression is characterized by development of plaque on the insides of arteries, which later hardens and narrows the arteries, leads to reduced supply of oxygen-rich blood to organs and other parts of the body. This can lead to various serious cardiovascular complications like heart attack, stroke, or even death (Paolo et al., 1995). Fig. 1 shows the development of atherosclerotic-plaque in the coronary artery.
Intracranial atherosclerosis
2014, The LancetCitation Excerpt :The disproportionately high rate of uncontrolled hypertension, diabetes mellitus, and hyperlipidaemia in the African-American population20,21 partly accounts for the increased incidence of intracranial atherosclerosis. The high rate in the Japanese population is attributed to the high frequency of hypertension with paradoxically low rates of hyperlipidaemia.22,23 In the Chinese population, the rates of hypertension, diabetes mellitus, and hyperlipidaemia are not higher than that in the white population,24–26 and therefore the excess burden of intracranial atherosclerosis is unaccounted for.
Urinary phosphate is associated with cardiovascular disease incidence
2023, Journal of Internal Medicine