Effect of daily variation in weather and sleep on seasonal affective disorder
References (16)
- et al.
Epidemiology and seasonal affective disorder
Psychological Medicine
(1988) Measurement of linear dependence and feedback between multiple time series
Journal of the American Statistical Association
(1982)A technique for the measurement of attitudes
Archives of General Psychology
(1932)Seasonal affective disorder, hibernation, and annual cycles in animals: Chipmunks in the sky
- Oren, D.A., and Rosenthal, N.E. Seasonal affective disorder. In: Paykel, E.S., ed. Handbook of Affective Disorders....
- Oren, D.A.; Shannon, N.J.; Carpenter, C.J.; and Rosenthal, N.E. Usage patterns of phototherapy in seasonal affective...
- et al.
Disturbance of the 24 hour rhythm in endogenous depression and the treatment of endogenous depression by sleep deprivation
International Pharmacopsychiatry
(1971) - et al.
Seasonal affective disorder: SAD or FAD?
Jefferson Journal of Psychiatry
(1989)
Cited by (33)
Hedging weather risk and coordinating supply chains
2018, Journal of Operations ManagementMeteorological analysis of symptom data for people with seasonal affective disorder
2017, Psychiatry ResearchCitation Excerpt :In a very small (n = 10) retrospective study it was found that the effect of the seasons on SAD is far more robust than the effects of the weather on energy and sleep patterns. The effects of the weather, if discernible, were most pronounced in the summer (Albert et al., 1991) or spring (Keller et al., 2005). In a larger study, no relationship between cloud cover, rainfall or atmospheric pressure and depression scores was found, but a significant relationship was found between depression scores of SAD patients and duration of sunshine, global radiation, temperature and length of daylight (Molin et al., 1996).
Particulate matter air pollution associated with hospital admissions for mental disorders: A time-series study in Beijing, China
2017, European PsychiatryCitation Excerpt :Usually, the nervous system responds first to the environmental variations, and the most common weather-triggered biological reactions often displayed a psychological, emotional or behavioral character [3]. Biophysical and meteorological factors may cause changes in the concentrations of cerebral neurotransmitters and eventually lead to mental or behavioral alterations [10,12,13]. Also, air pollution has recently been recognized as a suspected neurodevelopmental toxicant [14].
Weather, transport mode choices and emotional travel experiences
2016, Transportation Research Part A: Policy and PracticeCitation Excerpt :Other studies link moods directly to individual weather conditions. First, the number of sunshine hours and the level of solar radiation have been found to increase mood levels (e.g. Cunningham, 1979; Schwartz and Clore, 1983; Howarth and Hoffman, 1984; Barnston, 1988; Albert et al., 1991; Denissen et al., 2008; Ciucci et al., 2011). More specifically sunshine has been found to reduce sadness (Ciucci et al., 2011), cynicism, doubtfulness (Howarth and Hoffman, 1984), tiredness and sluggishness (Denissen et al., 2008), and increase optimism, concentration (Howarth and Hoffman, 1984) and mental activeness (Albert et al., 1991).
Assessing and hedging the cost of unseasonal weather: Case of the apparel sector
2015, European Journal of Operational ResearchReal estate prices: An international study of seasonality's sentiment effect
2012, Journal of Empirical FinanceCitation Excerpt :This hypothesis relies on the fact that numerous medical studies reveal that SAD affects the mood and health of a substantial portion of the population in the U.S., where the CDH is the main factor affecting SAD. In addition, there is also a SAD latitude effect above and beyond the effect of the CDH (see, e.g., Albert et al., 1991; Mersch et al., 1999). Indeed, controlling for the MT, BPH, and several important macroeconomic variables, we find that seasonality is highly significantly correlated with the SAD Onset/Recovery (OR) variable, empirically developed by Kamstra et al. (2009), and which reflects the change in the proportion of SAD-affected individuals actively suffering from SAD (as is further explained in the next section).
- 1
Paul S. Albert, Ph.D., is Staff Fellow, Biometry and Field Studies Branch, National Institute of Neurological Disorders and Stroke, Bethesda, MD.
- 2
Leora N. Rosen, Ph.D., is at the Uniformed Services University of the Health Sciences, Bethesda, MD.
- 3
Joseph R. Alexander, Jr., B.A., is in the Clinical Psychobiology Branch, National Institute of Mental Health, Bethesda, MD.
- 4
Norman E. Rosenthal, M.D., is Chief, Unit on Outpatient Studies, Clinical Psychobiology Branch, National Institute of Mental Health, Bethesda, MD.