Crystals and arthritis
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Cited by (61)
Dexamethasone causes calcium deposition and degeneration in human anterior cruciate ligament cells through endoplasmic reticulum stress
2020, Biochemical PharmacologyCitation Excerpt :Another study found that Dex treatment causes tendinous cells to form non-tendinous tissues (such as fat, chondral, and bone), making the tendon vulnerable to rupture [11]. Studies have shown that calcium deposition may be the latest event of ACL degeneration and that ACL calcification can even occur without cartilage calcification in some cases [36,46]. In this study, we found time- and concentration-independent increase in calcium deposition in human ACL cells treated with Dex, suggesting that the long-term effect of Dex causes an increase in calcium deposition in ACL cells.
Calcium Pyrophosphate Dihydrate Deposition Disease
2015, Mineral Scales and Deposits: Scientific and Technological ApproachesImaging in the crystal arthropathies
2014, Rheumatic Disease Clinics of North AmericaCitation Excerpt :These findings highlight the unique nature of the osteopathology of gout, which contrasts with that of immune-mediated arthropathies such as RA. CPPD crystal deposition disease is a common form of crystalline arthropathy characterized clinically by a variety of patterns including the most common asymptomatic state (radiographic chondrocalcinosis, Fig. 7), acute monoarthritis (pseudogout), and a symmetric polyarticular destructive arthropathy that mimics RA.56 A formal diagnosis of CPPD crystal deposition disease requires synovial fluid analysis and the finding of typical rhomboidal crystals with specific birefringence characteristics.
Endoscopic treatment of calcific tendinitis of the rectus femoris in a patient with intractable pain
2013, Journal of Orthopaedic ScienceAnakinra's Efficacy is Variable in Refractory Gout: Report of Ten Cases
2010, Seminars in Arthritis and RheumatismEffects of the physico-chemical nature of two biomimetic crystals on the innate immune response
2007, International ImmunopharmacologyCitation Excerpt :Polymethylmethacrylate (PMMA) particles generated by the breakdown of implanted prosthetics, such as the articulating surfaces of hip or knee replacements, migrate into various tissues and lead to activation of the host's inflammatory and immune responses [16]. In crystal deposition diseases such as arthritis and gout, it is the crystals of calcium pyrophosphate dihydrate (CPPD) or monosodium urate monohydrate (MSUM) that instigate painful acute inflammation after phagocytosis [15,17]. Basic calcium phosphate crystals (BCP, various admixtures of hydroxyapatite (HAP), octacalcium phosphate and tricalcium phosphate) are the particulates associated with the inflammation of osteoarthritis [18].