The electrocardiographic, clinical, and electrophysiologic spectrum of idiopathic monomorphic ventricular tachycardia☆
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Outflow Tract Ventricular Tachyarrhythmias: Mechanisms, Clinical Features, and Management
2018, Cardiac Electrophysiology: From Cell to Bedside: Seventh EditionExercise-induced Ventricular Tachycardia/Ventricular Fibrillation in the Normal Heart: Risk Stratification and Management
2016, Cardiac Electrophysiology ClinicsCitation Excerpt :Ventricular tachycardia (VT) and ventricular fibrillation (VF) occur only rarely in patients without organic heart disease, and most of these arrhythmic events are not related to exercise. Consequently, the literature on exercise-induced VT/VF in normal hearts is limited.1–3 Idiopathic exercise-induced VT is rare even in the young population, in which organic heart disease is less common.
Fascicular Ventricular Arrhythmias
2014, Cardiac Electrophysiology: From Cell to Bedside: Sixth EditionOutflow Tract Ventricular Tachyarrhythmias: Mechanisms, Clinical Features, and Management
2013, Cardiac Electrophysiology: From Cell to Bedside: Sixth EditionExtracorporeal membrane oxygenation support as bridge to recovery in a patient with electrical storm related cardiogenic shock
2013, American Journal of Emergency MedicineCitation Excerpt :One hour later, the patient's cardiac rhythm accelerated to 200 bpm, and she became hypotensive and hemodynamically unstable with mild renal insufficiency (creatinine: 94 μmol/L). The ECG showed a broad QRS tachycardia compatible with a VT according to classic criteria [2]. She was then transferred in the intensive care unit (ICU) after receiving 300 mg IV amiodarone and 50 mg oral (PO) metoprolol.
Ventricular Arrhythmias in Normal Hearts
2008, Cardiology ClinicsCitation Excerpt :β-blockers and calcium channel blockers may be used for chronic suppressive therapy; the efficacy in clinical studies has been variable, with success rates of up to 67% in patients who have typical RVOT tachycardia [46]. In some patients who have breakthrough tachycardia on β-blocker or calcium channel blockers, class I antiarrhythmic or class III antiarrhythmic therapy has been shown to be effective [1,47]. Although medical therapy may be effective in patients who have mild to moderate symptoms, it is frequently ineffective in patients who have severe symptoms [48].
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Supported by a grant from the Commisió Interdepartmental de Recerca i Innovació Tecnológia (Generalitat de Catalunya), Barcelona, Spain and by a grant from the “Sociedad Española de Cardiologia” Madrid, Spain.