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Causal Effect Heterogeneity

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Handbook of Causal Analysis for Social Research

Part of the book series: Handbooks of Sociology and Social Research ((HSSR))

Abstract

Individuals differ not only in background characteristics, often called “pretreatment heterogeneity,” but also in how they respond to a particular treatment, event, or intervention. A principal interaction of interest for questions of selection into treatment and causal inference in the social sciences is between the treatment and the propensity of treatment. Although the importance of “treatment-effect heterogeneity,” so defined, has been widely recognized in the causal inference literature, empirical quantitative social science research has not fully absorbed these lessons. In this chapter, we describe key estimation strategies for the study of heterogeneous treatment effects; we discuss recent research that attends to causal effect heterogeneity, with a focus on the study of effects of education, and what we gain from such attention; and we demonstrate the methods with an example of the effects of college on civic participation. The primary goal of this chapter is to encourage researchers to routinely examine treatment-effect heterogeneity with the same rigor they devote to pretreatment heterogeneity.

This research made use of facilities and resources at the California Center for Population Research, UCLA, which receives core support from the National Institute of Child Health and Human Development, Grant R24HD041022. Simon Thomas was supported by a UCLA Graduate Summer Research Mentorship program, the Institute for Research on Labor and Employment, and a pre-doctoral advanced quantitative methodology training grant (R305B080016) awarded to UCLA by the Institute of Education Sciences of the U.S. Department of Education. This research was conducted with restricted access to the Bureau of Labor Statistics (BLS) data. The views expressed here do not necessarily reflect the views of the BLS. We thank Yu Xie and Ben Jann for their contributions to related work, and Stephen Morgan and Judea Pearl for comments and suggestions. The ideas expressed herein are those of the authors.

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Notes

  1. 1.

    Extending to multivalued treatments with j values, the observed outcome variable contains only 1/J of the information in the potential outcome random variable, rather than ½ in the binary treatment setup. In other words, the proportion of unobservable counterfactual states increases as the number of treatment values j increases, such that we have a matrix of potential outcomes with j 2 cells, only the diagonal of which are observed (see Morgan and Winship 2007 for a detailed discussion).

  2. 2.

    Repeated observations of units of analysis can be used in fixed effects models to control for time-invariant unobserved properties of units, potentially increasing the plausibility of the assumption.

  3. 3.

    Heterogeneity in the effect of a binary endogenous regressor was introduced in the literature on switching regression models (Heckman 1978; Quandt 1972).

  4. 4.

    Bjorklund and Moffitt (1987) introduced the concept of the MTE and showed that the model was observationally equivalent to the switching regression model. See Carniero et al. (2011) for a description of related parameters, including the policy relevant treatment effect (PRTE) and the marginal policy relevant treatment effect (MPRTE). See Xie (2011) for a description of the incremental treatment effect (ITE), which is the average treatment effect for incremental units when a unit’s treatment status changes from d = 0 to d = 1 and when p increases from p 1 to p 2.

  5. 5.

    With a binary outcome, we use generalized methods of moments (Angrist 2001) or structural mean models or marginal structural models (Robins et al. 2000) rather than two-stage least squares.

  6. 6.

    Low inducement is a major limitation if the instrument is so weak as to have very little impact on the treatment of interest. We revisit this issue in the empirical example section.

  7. 7.

    We assume, for simplicity, that there are no “defiers,” that is, those individuals who would always do the opposite of treatment assignment.

  8. 8.

    See Zhou and Xie (2011) for further discussion of the difference between propensity score-based and IV approaches.

  9. 9.

    A recent study likewise suggests that economic returns to attending highly selective colleges are indistinguishable from zero among the full sample when adjustments for unobserved student characteristics are incorporated, while returns among Black and Hispanic students and students from disadvantaged families remain large (Dale and Krueger 2011). Hout (2012) reviews additional studies with corroborating results.

  10. 10.

    Brand and Davis (2011) combine the stratification-multilevel approach with discrete-time event-history models. Xie et al. (2011) also use the matching-smoothing and smoothing-differencing methods for the effects of college on fertility and find comparable results to those using stratification-multilevel. This is due to a largely linear pattern in effects of college on fertility across the propensity for college.

  11. 11.

    We impute missing values for our set of pretreatment covariates based on all other covariates. Most variables have 1–2% missing values. Only two variables are missing for more than 5% of the sample: parents’ income and high school college-preparatory program. We include an imputation indicator in our models.

  12. 12.

    Results controlling for the full set of covariates are very similar. Rosenbaum and Rubin (1983, 1984) demonstrate it is sufficient to condition on the propensity score as a function of X rather than X itself, which we do here for simplicity.

  13. 13.

    In contrast to Brand (2010), we impute all missing cases; the propensity score model specification also slightly differs from Brand (2010). Thus, our analyses yield marginally different results.

  14. 14.

    For the kth covariate in the jth stratum, we estimate the standardized mean covariate difference to quantify the balance between the treatment and the control groups for each covariate X (Morgan and Winship 2007):

    $$ {B_{k,j }}=\frac{{|{{\bar{X}}_{k,j,D=1 }}-{{\bar{X}}_{k,j,D=0 }}|}}{{\sqrt{{\frac{{S_{k,j,D=1}^2+S_{k,j,D=0}^2}}{2}}}}} $$

    where \( \bar{X} \) is the sample mean and S 2 is the sample variance of the kth covariate in the jth stratum for the treated and control groups as indexed by D = (1,0). The standardized difference is larger in some strata than in others for selected covariates.

  15. 15.

    We also fit alternative stratification-multilevel models in which we include quadratic terms in level-2. These terms were not statistically significant, and we do not present them here.

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Correspondence to Jennie E. Brand .

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Appendixes

Appendixes

Appendix A

Table 5 Descriptive statistics of pre-college covariates and civic participation (N = 3,452)

Appendix B

Table 6 Covariate and outcome means by propensity score strata and college completion (N = 3,452)

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Brand, J.E., Thomas, J.S. (2013). Causal Effect Heterogeneity. In: Morgan, S. (eds) Handbook of Causal Analysis for Social Research. Handbooks of Sociology and Social Research. Springer, Dordrecht. https://doi.org/10.1007/978-94-007-6094-3_11

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